Prolonged exposure to high fluoride levels during adolescence to adulthood elicits molecular, morphological, and functional impairments in the hippocampus

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Last updated 20 setembro 2024
Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
High Fluoride Levels May Affect Child Cognition - Neuroscience News
Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
Early‐life stress affects the structural and functional plasticity of the medial prefrontal cortex in adolescent rats - Chocyk - 2013 - European Journal of Neuroscience - Wiley Online Library
Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
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Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
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Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
Prolonged exposure to high fluoride levels during adolescence to adulthood elicits molecular, morphological, and functional impairments in the hippocampus
Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
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Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
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Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
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Prolonged exposure to high fluoride levels during adolescence to adulthood  elicits molecular, morphological, and functional impairments in the  hippocampus
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